Heart attack - Nursing Case Study
Pathophysiology
• Primary mechanism: Blockage of coronary arteries - A heart attack, or myocardial infarction, occurs when a coronary artery is blocked, often by a blood clot, reducing blood flow and oxygen to heart muscle cells, leading to cell death.
• Secondary mechanism: Plaque rupture - This blockage is usually due to atherosclerosis, where plaques made of fat, cholesterol, and other substances build up in the arteries. When these plaques rupture, they trigger clot formation, worsening the blockage.
• Key complication: Heart muscle damage - Reduced blood flow can cause permanent damage to heart muscle, impairing its ability to pump effectively, which may lead to heart failure or arrhythmias, affecting overall heart function and patient health.
Patient Profile
Demographics:
54 years old, male, office manager
History:
• Key past medical history: Hypertension diagnosed 5 years ago
• Current medications: Lisinopril 10 mg daily
• Allergies: No known drug allergies
Current Presentation:
• Chief complaint: Mild chest discomfort
• Key symptoms: Mild chest pain lasting 10 minutes, radiating to left arm, slight shortness of breath, no nausea or sweating
• Vital signs: Blood pressure 130/85 mmHg, heart rate 78 bpm, respiratory rate 16 breaths/min, temperature 98.6°F, oxygen saturation 98% on room air
Section 1
Initial Assessment Findings:
Upon admission to the emergency department, the nursing team conducted a thorough initial assessment of the patient. The patient reported that the mild chest discomfort had subsided, but he still experienced occasional episodes of slight shortness of breath. During the physical examination, the patient was alert and oriented, with skin that was warm and dry. Heart sounds were regular with no murmurs detected, and lung sounds were clear bilaterally. Peripheral pulses were palpable and symmetrical, and there was no jugular venous distension or peripheral edema noted.
An electrocardiogram (ECG) was performed, revealing ST-segment depression in the anterior leads, indicative of myocardial ischemia rather than a full-thickness infarction. Additionally, blood samples were taken for cardiac biomarkers, including troponin levels, to further assess for heart muscle damage. Given the patient's stable vital signs and the absence of severe symptoms, he was placed on continuous cardiac monitoring and started on low-dose aspirin to prevent further clot formation while awaiting additional diagnostic results. These findings suggest an early-stage myocardial infarction with a relatively stable clinical presentation, providing a foundation for continued monitoring and intervention to prevent progression of heart damage.
Section 2
New Diagnostic Results
As the patient remained under observation in the emergency department, the team received the results of the blood tests. The cardiac biomarkers showed mildly elevated troponin levels, consistent with myocardial ischemia, but not suggestive of significant myocardial damage. This confirmed the initial suspicion of an early-stage myocardial infarction, likely a Non-ST Elevation Myocardial Infarction (NSTEMI).
The patient's vital signs remained stable, with a blood pressure of 125/80 mmHg, heart rate of 78 beats per minute, respiratory rate of 18 breaths per minute, and oxygen saturation of 96% on room air. Given these encouraging findings, the medical team decided to start the patient on beta-blockers to reduce myocardial oxygen demand and statins to manage cholesterol levels. In addition, a low-molecular-weight heparin was administered to provide anticoagulation therapy.
These interventions were explained to the patient, who was reassured about the stable nature of his condition. The nursing team emphasized the importance of adhering to the medication regimen, monitoring for any signs of increased chest pain or shortness of breath, and maintaining a calm environment to reduce stress on the heart. The plan also included a referral for a cardiology consultation and a non-invasive stress test to further evaluate the patient's cardiac function before considering discharge. This approach aimed to ensure comprehensive care and prevent any potential complications.
Section 3
Response to Interventions
Over the next several hours, the patient's response to the initial interventions was closely monitored by the nursing team. The patient remained stable, with vital signs consistently within normal ranges: blood pressure at 122/78 mmHg, heart rate at 76 beats per minute, respiratory rate at 18 breaths per minute, and oxygen saturation maintaining at 97% on room air. The patient reported a mild decrease in the sensation of chest pressure, which was a reassuring sign of the effectiveness of the prescribed medications. He was cooperative and appeared comfortable, expressing understanding and compliance with the treatment plan.
Nurses conducted regular assessments, focusing on the patient's pain levels, respiratory status, and overall comfort. They noted an absence of any new or worsening symptoms, such as increased chest pain, shortness of breath, or diaphoresis. The patient was encouraged to rest but also advised on the importance of early mobilization, as tolerated, to prevent complications such as deep vein thrombosis. Additionally, the patient was educated on recognizing signs that would necessitate immediate medical attention, including significant chest pain, difficulty breathing, or feelings of faintness.
As the shift progressed, the nursing team prepared the patient for the scheduled cardiology consultation and non-invasive stress test, ensuring he was informed and at ease about the upcoming procedures. The team reinforced the importance of lifestyle modifications, including diet and activity changes, to support cardiac health. With a stable condition and positive response to treatment, the patient was on track for further evaluation and eventual discharge planning, fostering confidence in his recovery process.
Section 4
As the patient's condition remained stable, the nursing team received new diagnostic results that provided further insight into his cardiac health. The results from the non-invasive stress test showed mild ischemic changes, indicating areas of the heart muscle that were not receiving adequate blood flow under stress. However, these changes were consistent with an early-stage cardiac event and did not suggest significant obstruction requiring immediate intervention. The patient's blood work confirmed normal levels of cardiac enzymes, ruling out ongoing myocardial damage. Lipid profile results revealed elevated LDL cholesterol levels, highlighting an area of focus for long-term management.
The nursing team discussed these findings with the patient, emphasizing the importance of adhering to the prescribed medication regimen, which included antiplatelet therapy and statins, to manage cholesterol levels and reduce the risk of future cardiac events. The patient expressed a willingness to make necessary lifestyle changes, including dietary adjustments and a gradual increase in physical activity, as recommended by the cardiologist. Nurses provided educational materials and set achievable goals to empower the patient in his recovery journey.
In preparation for discharge, the nursing team collaborated with the interdisciplinary team to develop a comprehensive care plan. This plan included follow-up appointments with the cardiologist, a referral to a cardiac rehabilitation program, and ongoing monitoring of vital signs and symptoms. The patient was instructed on the use of a home blood pressure monitor and encouraged to maintain a daily log of his readings. With a clear understanding of his condition and the steps needed to promote his heart health, the patient felt confident and supported as he prepared to transition from hospital care to managing his health at home.
Section 5
As the patient progressed with his recovery at home, he diligently adhered to the prescribed medication regimen and lifestyle changes. During a routine follow-up appointment with the cardiologist, new diagnostic tests were conducted to assess his ongoing cardiac health. An echocardiogram revealed that while the overall heart function remained stable, there was evidence of mild left ventricular hypertrophy, likely attributable to longstanding hypertension. This finding prompted the team to reassess the patient's blood pressure management strategy to ensure optimal control.
The patient's vital signs indicated a blood pressure of 138/85 mmHg, slightly above the target range, and a heart rate of 78 beats per minute. Given these numbers, the cardiologist, in collaboration with the nursing team, adjusted the patient's antihypertensive medication to better manage his blood pressure and prevent further cardiac strain. Additionally, the patient's lipid profile showed some improvement, with LDL cholesterol levels reduced from the previous measurement, indicating a positive response to the statin therapy.
To address the mild hypertrophy and to further reinforce heart-healthy habits, the nursing team provided the patient with additional educational resources. These resources emphasized the importance of maintaining a low-sodium diet, managing stress, and incorporating regular, moderate exercise into his daily routine. The patient was encouraged to continue monitoring his blood pressure at home and to stay engaged in the cardiac rehabilitation program, which had been instrumental in guiding his gradual increase in physical activity. With these adjustments and ongoing support, the patient remained optimistic about his continued recovery and cardiovascular health.