fracture - Nursing Case Study
Pathophysiology
• Primary mechanism: A fracture occurs when the bone is subjected to stress exceeding its strength, leading to a break. This stress can result from acute trauma, repetitive force (stress fracture), or weakened bone structure due to conditions like osteoporosis.
• Secondary mechanism: The fracture disrupts blood vessels within the bone and surrounding tissues, initiating an inflammatory response. This response involves the release of cytokines and growth factors, promoting the formation of a hematoma and the recruitment of cells necessary for bone healing.
• Key complication: If not properly managed, fractures can lead to malunion or nonunion, where the bone heals improperly or fails to heal, respectively. This can result in chronic pain, deformity, and impaired function, highlighting the importance of appropriate fracture management and rehabilitation.
Patient Profile
Demographics:
45-year-old male, construction worker
History:
• Key past medical history: Hypertension, type 2 diabetes
• Current medications: Lisinopril, Metformin
• Allergies: Penicillin
Current Presentation:
• Chief complaint: Severe pain in the left leg after a fall from a scaffolding
• Key symptoms: Swelling and bruising around the left thigh, inability to bear weight, tingling sensation in the left foot
• Vital signs: Blood pressure 150/95 mmHg, heart rate 110 bpm, respiratory rate 22 breaths per minute, temperature 99.1°F
Section 1
During the initial assessment, the patient was found to have a closed mid-shaft femur fracture in his left leg, confirmed by X-ray imaging. The fracture was comminuted, indicating multiple bone fragments. The significant swelling and bruising suggested a possible compartment syndrome, a condition where increased pressure within the muscle compartments could compromise circulation and lead to tissue damage. The tingling sensation in the left foot raised concerns about potential nerve involvement or vascular compromise. Given the patient's history of hypertension and diabetes, there was an elevated risk for delayed healing and infection, necessitating vigilant monitoring and management.
The patient's vital signs showed hypertension and tachycardia, which were likely stress responses to pain and injury. However, these signs could also suggest inadequate perfusion or developing shock, especially if the fracture had led to significant internal bleeding. Laboratory tests revealed a hemoglobin level of 10.5 g/dL, indicating mild anemia, possibly due to bleeding from the fracture site. The blood glucose level was elevated at 250 mg/dL, which could impede healing and increase the risk for infection.
The initial intervention focused on stabilizing the fracture with traction to prevent further damage and alleviate pain, along with aggressive pain management and monitoring for signs of compartment syndrome. Despite these efforts, the patient began to demonstrate increasing pain and swelling, alongside a drop in blood pressure to 135/90 mmHg and a further increase in heart rate to 120 bpm. These changes suggested the possibility of a developing complication, such as compartment syndrome or hemorrhagic shock, prompting the need for urgent reevaluation and potential surgical intervention to decompress the affected compartment and stabilize the fracture site.
Section 2
As the medical team proceeded with the ongoing assessment, the patient's condition raised considerable concern. The increasing pain and swelling in his left leg, coupled with the persistent tingling sensation, necessitated an immediate re-evaluation for compartment syndrome. To confirm this suspicion, intracompartmental pressure measurements were obtained, revealing pressures exceeding 30 mmHg, which strongly indicated the presence of compartment syndrome. This critical finding required prompt surgical intervention in the form of a fasciotomy to relieve the pressure and prevent irreversible muscle and nerve damage.
Simultaneously, the patient's vital signs continued to reflect instability. The heart rate persistently remained elevated at 120 bpm, and blood pressure showed a further decline to 125/85 mmHg. These changes, along with the mild anemia and ongoing blood loss, raised the possibility of hemorrhagic shock. Given the comminuted nature of the fracture, there was concern for ongoing internal bleeding. A repeat hemoglobin test showed a further drop to 9.8 g/dL, and coagulation profiles were ordered to assess the bleeding risk. The team decided to transfuse packed red blood cells to improve oxygen carrying capacity and stabilize hemodynamics while preparing for potential surgical intervention.
The elevated blood glucose level of 250 mg/dL persisted, despite initial insulin administration, emphasizing the need for tighter glucose control to mitigate infection risks and promote healing. The patient's history of diabetes heightened the vigilance for infection, particularly with the impending surgical fasciotomy. Plans were made to increase the frequency of glucose monitoring and adjust insulin therapy accordingly. The clinical reasoning now focused on balancing the urgent need for surgical intervention with careful management of the patient's comorbid conditions, aiming to stabilize his overall condition and prevent further complications in the post-operative phase.
Section 3
As the medical team proceeded with the fasciotomy, attention was carefully directed towards managing the patient's evolving clinical picture. Post-operatively, the focus shifted to monitoring for signs of infection and ensuring effective pain control. The initial hours following the fasciotomy were marked by a slight improvement in the patient’s pain levels and a reduction in swelling, suggesting a successful pressure release. However, careful surveillance for signs of infection was critical given the patient's diabetes and elevated glucose levels. The surgical site was kept clean and dry, and a broad-spectrum antibiotic was initiated prophylactically to mitigate infection risk.
Despite these efforts, the patient's condition showed signs of new complications two days post-surgery. The wound site began to exhibit erythema and warmth, and the patient developed a low-grade fever of 100.4°F. These findings, coupled with an increase in white blood cell count to 14,000/mm³, raised concerns for a developing wound infection. Additionally, the patient reported increased fatigue and malaise, which could be attributed to the systemic inflammatory response. Blood cultures were obtained to identify any causative organisms, and antibiotic therapy was adjusted based on preliminary sensitivity profiles.
In conjunction with the infectious concerns, the patient's respiratory status began to decline, evidenced by an increased respiratory rate of 24 breaths per minute and a drop in oxygen saturation to 92% on room air. Given the patient's history and recent surgical intervention, the possibility of a pulmonary embolism was considered. A CT pulmonary angiogram was ordered to explore this suspicion further, while supplemental oxygen was provided to maintain adequate oxygenation. As the team awaited diagnostic results, clinical reasoning centered on the delicate balance between managing the patient’s acute complications and addressing his chronic conditions, ensuring a comprehensive approach to his care and recovery.
Section 4
The CT pulmonary angiogram returned positive for a small pulmonary embolism in the right lower lobe, confirming the team's suspicions. Given the patient's increased respiratory rate and decreased oxygen saturation, the primary focus shifted to managing this acute complication while continuing to monitor signs of infection at the fasciotomy site. Anticoagulation therapy was promptly initiated with low molecular weight heparin to address the embolism, balancing the risk of bleeding against the need to prevent further thromboembolic events. The patient's oxygen supplementation was adjusted to maintain saturation levels above 94%, and respiratory therapy was consulted to optimize pulmonary function and support.
Meanwhile, the wound culture results indicated the presence of methicillin-resistant Staphylococcus aureus (MRSA), necessitating a change in antibiotic regimen to vancomycin, tailored to the sensitivity profile. The patient's low-grade fever persisted, and inflammatory markers such as C-reactive protein (CRP) and erythrocyte sedimentation rate (ESR) were elevated, suggesting ongoing infection and systemic inflammation. The team coordinated an interdisciplinary approach involving infectious disease specialists to refine the antibiotic strategy and ensure effective infection control measures.
Throughout these developments, the nursing team played a crucial role in continuous assessment and patient education. They emphasized the importance of glycemic control to enhance wound healing and reduce infection risk, considering the patient's diabetes. Nursing interventions included regular monitoring of blood glucose levels and insulin adjustments as necessary. The patient's status was communicated effectively during team rounds, highlighting the interconnectedness of the acute pulmonary issue and the chronic wound management, ensuring a holistic approach to care. These steps were critical in stabilizing the patient and setting the stage for potential discharge planning once the acute issues were managed and controlled.
Section 5
As the patient's care progressed, a new complication emerged. Over the course of several days, the patient began experiencing increased swelling and pain in the affected limb, despite adherence to the prescribed anticoagulation and antibiotic therapies. The nursing assessment revealed that the limb was warmer to the touch with a significant increase in circumference compared to previous measurements, suggesting potential deep vein thrombosis (DVT) or worsening of the existing inflammation. The patient's vital signs indicated a slight increase in heart rate to 102 beats per minute, with blood pressure remaining stable at 135/85 mmHg. The oxygen saturation was maintained at 95% with supplemental oxygen. Laboratory results indicated a further elevation in D-dimer levels, prompting the team to order a duplex ultrasound to evaluate for DVT.
The ultrasound confirmed the presence of a new DVT in the popliteal vein, complicating the patient's clinical picture. This finding necessitated a reassessment of the current anticoagulation strategy, weighing the benefits and risks of intensifying therapy. The healthcare team, including a hematologist, was consulted to optimize anticoagulation management, considering the dual diagnosis of pulmonary embolism and DVT. Meanwhile, the infectious disease team continued to monitor the MRSA infection closely, ensuring that vancomycin levels were therapeutic and adjusting the regimen as needed based on the patient's renal function.
The nursing staff focused on patient education regarding signs and symptoms of potential complications, emphasizing the importance of reporting any sudden changes in limb sensation or increased shortness of breath. They also reinforced the need for consistent limb elevation and encouraged gentle mobility to promote venous return. Despite the new challenges, the patient's overall condition remained stable, with a plan to reassess the therapeutic approach as the clinical picture evolved. This careful monitoring and coordinated care aimed to address both the thromboembolic and infectious complications, setting the groundwork for the patient's eventual recovery and discharge planning.