stroke - Nursing Case Study

Pathophysiology

• Primary mechanism: Ischemic stroke occurs when a blood clot obstructs a cerebral artery, leading to reduced blood flow and oxygen to brain tissue, causing cell injury and death.

• Secondary mechanism: Hemorrhagic stroke results from the rupture of a blood vessel in the brain, leading to bleeding, increased intracranial pressure, and subsequent neuronal damage.

• Key complication: Both types can cause localized brain damage, resulting in neurological deficits such as paralysis, speech difficulties, and cognitive impairment, which are crucial for understanding patient care and rehabilitation strategies.

Patient Profile

Demographics:

65-year-old female, retired school teacher

History:

• Key past medical history: Hypertension, Type 2 Diabetes, Hyperlipidemia

• Current medications: Lisinopril, Metformin, Atorvastatin, Aspirin

• Allergies: Penicillin

Current Presentation:

• Chief complaint: Sudden weakness on the right side of the body

• Key symptoms: Slurred speech, facial droop on the right side, difficulty walking, mild headache

• Vital signs: Blood Pressure: 180/100 mmHg, Heart Rate: 95 bpm, Respiratory Rate: 22 breaths/min, Temperature: 98.6°F, Oxygen Saturation: 93% on room air

Section 1

As the medical team initiates the initial assessment, the patient, Mrs. Thompson, undergoes a detailed neurological evaluation and imaging studies. A CT scan of the head is performed promptly, revealing an ischemic stroke in the left middle cerebral artery territory. The stroke team is consulted immediately, and tissue plasminogen activator (tPA) is considered within the treatment window to dissolve the clot. Blood glucose levels are assessed given her history of Type 2 Diabetes, showing a level of 160 mg/dL, slightly elevated but not critically high. The team ensures her glucose is managed to prevent further neurological damage. Her blood pressure remains elevated at 185/98 mmHg, a critical factor that requires careful, gradual reduction to avoid further cerebral perfusion issues.

Shortly after the initial intervention, Mrs. Thompson's condition shows signs of change. Her oxygen saturation drops to 89% on room air, prompting supplemental oxygen administration via nasal cannula. Her respiratory rate increases to 26 breaths per minute, indicating possible respiratory compromise. Auscultation of lung sounds reveals fine crackles at the bases, suggesting the development of pulmonary congestion, potentially secondary to heart strain or aspiration risk due to dysphagia associated with her stroke. The nursing team implements aspiration precautions, elevating the head of the bed and preparing for a possible swallow evaluation.

These developments necessitate a coordinated response from the healthcare team. The primary focus is on stabilizing Mrs. Thompson's cardiovascular and respiratory status while monitoring for signs of neurological deterioration. The team establishes a plan for frequent vital sign monitoring, neurological checks, and strict blood glucose control. Mrs. Thompson's case highlights the complexity of stroke management, requiring astute clinical reasoning as her condition evolves. The team remains vigilant for potential new complications, including cardiac events and infection risks, as they adjust her care plan to optimize recovery and prevent further deterioration.

Section 2

As the healthcare team continues to monitor Mrs. Thompson's condition, they note a change in her neurological status. During a routine neurological check, Mrs. Thompson exhibits increased confusion and difficulty speaking, with her speech becoming garbled and less coherent than during the initial assessment. Her right-sided weakness appears more pronounced, and she is unable to lift her right arm against gravity. These findings raise concerns about potential expansion of the ischemic stroke or new complications, such as hemorrhagic transformation. The nurse immediately reports these observations to the stroke team, prompting an urgent repeat CT scan to assess for any changes in cerebral pathology.

The repeat CT scan reveals no evidence of hemorrhagic transformation; however, it does show mild cerebral edema around the ischemic area, contributing to her altered mental status. This finding necessitates careful management to reduce intracranial pressure while maintaining adequate cerebral perfusion. The medical team considers the administration of osmotic diuretics such as mannitol and elevates the head of the bed to 30 degrees to optimize venous drainage. Additionally, the team continues to monitor her blood pressure closely to ensure it remains at a level that supports adequate cerebral perfusion without exacerbating the edema.

In parallel, Mrs. Thompson's blood glucose levels are monitored rigorously, as hyperglycemia could worsen her neurological status. The nursing team administers insulin as needed to maintain her glucose levels within a target range. As the team implements these interventions, they remain vigilant for signs of further deterioration or new complications, such as aspiration pneumonia, given her increased risk due to dysphagia. The focus is on stabilizing Mrs. Thompson's condition and optimizing her recovery through coordinated, multidisciplinary care tailored to her evolving clinical needs. This phase of her treatment underscores the importance of dynamic clinical reasoning and timely intervention in managing complex stroke cases.

Section 3

As the healthcare team continues to monitor Mrs. Thompson closely, her response to the interventions implemented for cerebral edema begins to unfold. Over the next several hours, the nursing team observes a gradual improvement in her neurological status. Her speech, while still impaired, becomes slightly more coherent, and she exhibits a mild reduction in confusion. Her right-sided weakness remains, but she is now able to move her right arm slightly against gravity, suggesting a positive response to the current management strategy. Vital signs are stable, with her blood pressure maintained at 140/85 mmHg, supporting adequate cerebral perfusion.

However, despite these encouraging signs, Mrs. Thompson begins to develop a low-grade fever, with her temperature rising to 38.2°C (100.8°F). This prompts the team to investigate potential sources of infection, given her increased risk for aspiration pneumonia due to her dysphagia. A chest X-ray is ordered to assess for any signs of pulmonary complications. Additionally, the nursing team performs a comprehensive assessment, including auscultation of lung sounds, which reveal slight crackles in the right lower lobe, raising suspicion of potential aspiration.

Laboratory results reveal a white blood cell count of 13,000/mm³, further supporting the possibility of infection. The team initiates empiric antibiotic therapy while awaiting culture results, recognizing the importance of early intervention in preventing further deterioration. This new development underscores the complexity of Mrs. Thompson's case, as the team balances the management of her stroke-related complications with the emerging need to address potential infectious processes. The multidisciplinary team remains vigilant, adjusting the care plan as needed to optimize her recovery and prevent further setbacks.

Section 4

As the healthcare team continues to monitor Mrs. Thompson, they note a change in her respiratory status. Her respiratory rate increases to 24 breaths per minute, and she appears to be working harder to breathe. Oxygen saturation levels have decreased slightly, now reading at 91% on room air. Given these changes, the team decides to initiate supplemental oxygen therapy to maintain adequate oxygenation, starting with a nasal cannula at 2 liters per minute and monitoring her response closely. The crackles in her right lower lobe persist, and the team considers the possibility of evolving pneumonia, likely secondary to aspiration, as the primary cause of her respiratory compromise.

Concurrent with these respiratory observations, the results of Mrs. Thompson's chest X-ray are made available. The imaging confirms the presence of an infiltrate in her right lower lobe, consistent with aspiration pneumonia. This finding corroborates the earlier clinical suspicion and guides the refinement of her antibiotic regimen to target the most likely pathogens effectively. The healthcare team also emphasizes the importance of preventative measures, such as elevating the head of her bed and implementing meticulous oral care to reduce further aspiration risks.

Despite these setbacks, Mrs. Thompson continues to show improvement in her neurological status. Her speech becomes increasingly coherent, and she demonstrates the ability to follow simple commands. This simultaneous progression in her neurological recovery and development of mild respiratory complications presents a complex clinical picture requiring careful prioritization and adjustment of her care plan. The team remains proactive, maintaining a delicate balance between addressing her current respiratory needs and supporting her ongoing recovery from the stroke. As they await culture results to further tailor her treatment, the focus remains on preventing additional complications and promoting Mrs. Thompson's overall recovery trajectory.

Section 5

As the healthcare team continues to monitor Mrs. Thompson, they observe a new complication in her clinical status. Over the course of the next 24 hours, Mrs. Thompson's respiratory effort remains labored despite the supplemental oxygen, and her oxygen saturation levels fluctuate, occasionally dipping to 89% on 2 liters per minute via nasal cannula. Her respiratory rate increases further to 28 breaths per minute, and she exhibits signs of accessory muscle use, indicating increased respiratory distress. Additionally, Mrs. Thompson begins to exhibit a low-grade fever, with her body temperature rising to 100.4°F (38°C), further suggesting an inflammatory or infectious process.

In parallel, the laboratory results from her blood work reveal a slight increase in her white blood cell count to 12,500/mm³, supporting the diagnosis of an infection, likely related to her confirmed aspiration pneumonia. The team also notes a mild elevation in her C-reactive protein (CRP) levels, signaling an acute inflammatory response. Given these findings, the healthcare team decides to escalate her antibiotic therapy to a broader spectrum regimen while awaiting specific culture results to ensure coverage of possible resistant organisms. Additionally, they implement more aggressive pulmonary hygiene measures, including physiotherapy and incentive spirometry, to aid in clearing her lung secretions and enhance her respiratory function.

Throughout this period, Mrs. Thompson's neurological status continues to improve, though her respiratory compromise poses a significant challenge. Her ability to communicate and follow commands remains a positive indicator of her stroke recovery, but the team must maintain vigilance to prevent further deterioration in her respiratory status. The interdisciplinary team collaborates closely, ensuring that each intervention is carefully balanced to support Mrs. Thompson's overall recovery journey, emphasizing the importance of ongoing assessment and timely adjustment of her treatment plan to accommodate her evolving clinical needs.