Left femur fracture - Nursing Case Study

Pathophysiology

• Primary mechanism: Disruption of bone integrity occurs due to a direct impact or excessive force, causing the femur to fracture. This results in displacement or angulation of bone fragments, impairing normal limb function and stability.

• Secondary mechanism: Vascular injury may accompany the fracture, leading to potential bleeding and hematoma formation. This can compromise blood supply to surrounding tissues, increasing the risk of avascular necrosis if not promptly managed.

• Key complication: Improper or delayed treatment can lead to malunion or nonunion of the fracture, potentially causing chronic pain, reduced mobility, and long-term disability. Early intervention and stabilization are crucial to prevent these outcomes.

Patient Profile

Demographics:

45-year-old male, construction worker

History:

• Key past medical history: Hypertension, Type 2 Diabetes

• Current medications: Lisinopril, Metformin

• Allergies: Penicillin

Current Presentation:

• Chief complaint: Severe pain and swelling in the left thigh

• Key symptoms: Inability to bear weight on the left leg, bruising and deformity around the left thigh, increasing pain despite pain medication

• Vital signs: Blood pressure 150/95 mmHg, heart rate 110 bpm, respiratory rate 22 breaths per minute, temperature 38.2°C (100.8°F), oxygen saturation 95% on room air

Section 1

As the clinical team continues to monitor the 45-year-old construction worker with a left femur fracture, new complications begin to emerge. Despite initial stabilization efforts, the patient exhibits signs of worsening vascular compromise in the affected limb. Upon further assessment, the left leg appears increasingly pale and cool to the touch, with capillary refill time extending beyond 3 seconds, indicating potential impaired perfusion. The patient's pain also escalates to an excruciating level despite maximal non-opioid analgesia, raising concerns about compartment syndrome, a serious condition that can result from increased pressure within the muscle compartments of the leg.

New diagnostic results, including a Doppler ultrasound, reveal diminished arterial flow distal to the fracture site, confirming significant vascular involvement. Additionally, laboratory tests show an elevated white blood cell count of 14,500/mm³, suggesting an inflammatory response, possibly due to tissue injury or an emerging infection. These findings prompt the medical team to consider urgent surgical intervention to relieve pressure and restore adequate blood flow to the limb, thus preventing further tissue damage and potential loss of limb function.

The patient's status necessitates a multidisciplinary approach, with input from orthopedic surgeons, vascular specialists, and the nursing team to optimize care. The priority is to address both the fracture and the vascular compromise, recognizing that timely intervention is critical to prevent irreversible muscle and nerve damage. This evolving scenario underscores the importance of vigilant monitoring and proactive management to mitigate the risk of long-term complications such as chronic pain or disability, guiding the next steps in the patient's treatment journey.

Section 2

As the clinical team proceeds with the plan for surgical intervention, they prioritize the patient's stabilization and closely monitor for any further complications. The patient's vital signs reveal a heart rate of 110 beats per minute, a blood pressure of 98/68 mmHg, and a respiratory rate of 22 breaths per minute, indicating a compensatory response likely due to pain and stress. Despite these measures, the patient begins to exhibit signs of systemic involvement. Notably, there is an onset of mild tachypnea and intermittent confusion, suggesting the possibility of systemic inflammatory response syndrome (SIRS) secondary to either the trauma or a developing infection.

In response to these changes, the medical team orders additional laboratory tests, including a complete blood count and blood cultures, to assess for potential bacteremia. The blood cultures return positive for Staphylococcus aureus, raising the concern of a possible osteomyelitis or wound infection, which could further complicate the patient's condition. The elevated white blood cell count, now at 16,200/mm³, corroborates the suspicion of an infectious process. Immediate initiation of broad-spectrum intravenous antibiotics is undertaken, tailored to the suspected pathogen, to prevent further systemic spread and complications.

These developments necessitate a reevaluation of the surgical plan to incorporate debridement and possible drainage alongside fracture stabilization and vascular repair. The interdisciplinary team discusses the need to adjust the patient's care plan accordingly, emphasizing the importance of addressing both the infectious and vascular components swiftly. This coordinated approach aims to reduce the risk of sepsis, preserve limb function, and promote optimal recovery, highlighting the critical role of timely and comprehensive management in mitigating the potential for long-term disability.

Section 3

As the medical team continues to manage the patient's condition, they closely monitor the response to the initiated antibiotic therapy. Within 48 hours, the patient's vital signs show slight improvement, with a heart rate of 102 beats per minute, a blood pressure of 104/70 mmHg, and a respiratory rate of 20 breaths per minute, indicating a potential stabilization of the systemic inflammatory response. However, the patient still experiences intermittent confusion, and new laboratory results reveal a persistent elevation in inflammatory markers, including a C-reactive protein level of 12 mg/L and an erythrocyte sedimentation rate of 45 mm/hr. These findings underscore the need for vigilant monitoring and evaluation of the underlying infection and inflammatory state.

Despite the initial positive response to antibiotics, further assessment reveals localized swelling and redness around the surgical site, with purulent drainage noted upon inspection. This development suggests a possible wound infection, potentially complicating the recovery process and necessitating immediate intervention to prevent further deterioration. The surgical team decides to perform an urgent debridement to remove necrotic tissue and drain any accumulated pus, which is essential to control the spread of infection and promote healing.

In light of these findings, the clinical team emphasizes the importance of a multidisciplinary approach, incorporating infectious disease specialists and wound care nurses to optimize treatment strategies. They also adjust the patient's antibiotic regimen based on culture sensitivities to ensure targeted microbial eradication. This comprehensive approach is critical to addressing both the infectious complications and ensuring successful fracture stabilization, ultimately aiming to preserve limb function and enhance the patient's overall recovery trajectory. As the patient progresses through this critical phase, the team remains vigilant for any signs of new complications, prepared to adapt the care plan to meet evolving clinical needs.

Section 4

A few days post-debridement, the patient's condition takes a concerning turn, illustrating a new complication in the recovery process. Despite the initial improvement in systemic signs, the patient now shows signs of acute respiratory distress. Upon assessment, the patient exhibits increased respiratory rate at 28 breaths per minute, oxygen saturation levels have dropped to 88% on room air, and lung auscultation reveals diminished breath sounds with crackles in the lower lobes bilaterally. The patient's confusion has worsened, and she is intermittently disoriented, which raises suspicion for potential hypoxia or other systemic involvement.

New diagnostic results are prioritized to identify the underlying cause of these respiratory changes. A chest X-ray reveals bilateral infiltrates, suggesting the development of hospital-acquired pneumonia, possibly secondary to prolonged immobilization and compromised respiratory function. Blood cultures are drawn again to rule out secondary bacteremia, and arterial blood gases indicate a pH of 7.31, PaCO2 of 48 mmHg, and PaO2 of 60 mmHg, confirming respiratory acidosis with hypoxemia. These findings necessitate a re-evaluation of the current therapeutic plan and highlight the importance of respiratory support and infection control.

In response to these developments, the clinical team implements an immediate intervention plan. The patient is started on supplemental oxygen via nasal cannula, with plans to escalate to non-invasive ventilation if her respiratory status does not improve. Empiric broad-spectrum antibiotics are initiated while awaiting culture results to address the potential pneumonia, and the respiratory therapy team is engaged to optimize pulmonary hygiene and facilitate secretion clearance. The multidisciplinary team remains focused on addressing this new complication, with an eye toward preventing further escalation of the patient's respiratory compromise and ensuring comprehensive care that supports her recovery from both the fracture and the infectious challenges.

Section 5

Response to interventions:

Following the initiation of supplemental oxygen therapy via nasal cannula at 4 liters per minute, the patient's oxygen saturation improves slightly to 92%, but her respiratory rate remains elevated at 26 breaths per minute. Despite the introduction of empiric broad-spectrum antibiotics, the patient's fever spikes to 101.5°F, suggesting ongoing infectious processes. The patient's mental status, however, shows modest improvement; she is now more alert and oriented to person and place, although she still struggles with recall of recent events.

The respiratory therapy team implements chest physiotherapy and instructs the patient in incentive spirometry usage to enhance lung expansion and secretion clearance. With these interventions, the patient demonstrates increased expectoration of purulent sputum. Repeat arterial blood gases show a slight improvement in pH to 7.35, PaCO2 reduced to 45 mmHg, and a PaO2 of 64 mmHg, indicating partial improvement in respiratory acidosis and hypoxemia. These changes suggest a positive response to the combined respiratory and antibiotic therapies, though careful monitoring remains essential.

However, the team remains vigilant for further complications. Given the patient's prolonged immobility and recent surgical history, there is a heightened risk for thromboembolic events. Prophylactic anticoagulation is considered to mitigate this risk, and the patient's fluid balance is closely monitored to prevent fluid overload, which could exacerbate respiratory compromise. The clinical team continues to assess the need for escalating respiratory support, particularly if the patient's respiratory parameters do not stabilize or if her oxygen requirements increase. This ongoing assessment underscores the need for a dynamic and responsive care plan tailored to address the evolving complexity of her condition.