failure to thrive - Nursing Case Study
Pathophysiology
• Primary mechanism: Inadequate caloric intake – Insufficient nutritional provision or absorption leads to energy deficits, impacting growth and development. This can result from feeding difficulties, socio-economic factors, or gastrointestinal disorders.
• Secondary mechanism: Increased metabolic demands – Conditions such as chronic infections or congenital heart defects elevate metabolic rates, requiring more calories than consumed, exacerbating growth failure.
• Key complication: Impaired growth and development – Persistent energy deficits hinder physical and cognitive development, potentially leading to long-term health issues and developmental delays.
Patient Profile
Demographics:
2-year-old female, daycare attendee
History:
• Key past medical history: Born at 36 weeks, history of GERD (gastroesophageal reflux disease)
• Current medications: Lansoprazole
• Allergies: No known drug allergies
Current Presentation:
• Chief complaint: Poor weight gain and delayed developmental milestones
• Key symptoms: Lethargy, decreased appetite, irritability, occasional vomiting
• Vital signs: Temperature 99.1°F, Heart rate 130 bpm, Respiratory rate 30 breaths/min, Blood pressure 85/50 mmHg
Section 1
As the healthcare team continued to monitor the 2-year-old female, her initial assessment findings revealed additional concerns that warranted further investigation. During a thorough physical examination, the pediatric nurse noted that the child exhibited signs of muscle wasting and a protruding abdomen, indicative of possible malnutrition. Her skin appeared pale and slightly dry, and there was minimal subcutaneous fat. The child's developmental assessment indicated delays in both gross motor skills and speech, as she struggled to walk without support and could articulate only a few simple words. Her parents reported that she seemed less interested in play and social interaction than her peers at daycare, further supporting concerns about her developmental progress.
Laboratory tests were ordered to gain more insight into the underlying causes of her failure to thrive. The results showed mild anemia with a hemoglobin level of 10.5 g/dL and a hematocrit of 32%, suggesting iron deficiency. Serum albumin levels were slightly reduced at 3.4 g/dL, pointing toward inadequate protein intake or absorption. Additionally, a comprehensive metabolic panel revealed slightly elevated liver enzymes, raising concerns about potential liver involvement or chronic malabsorption issues. Stool analysis was conducted to check for fat malabsorption and possible parasitic infections, with initial findings indicating steatorrhea, further hinting at a possible malabsorption syndrome such as celiac disease or another gastrointestinal disorder.
These findings prompted the healthcare team to adjust their approach, considering both nutritional and potential gastrointestinal interventions. A nutritionist was consulted to develop a tailored dietary plan aimed at addressing her caloric and micronutrient deficiencies. Meanwhile, referrals to a pediatric gastroenterologist and a developmental specialist were initiated to explore potential gastrointestinal causes and provide targeted support for her developmental delays. The team recognized the complexity of the case and the need for a multidisciplinary approach to ensure comprehensive care, laying the groundwork for the next steps in the child’s management and ongoing assessment.
Section 2
As the healthcare team continued to address the child's condition, new diagnostic results shed light on her complex medical picture. The pediatric gastroenterologist conducted a series of tests, including a tissue transglutaminase antibody test, which returned positive, suggesting a strong possibility of celiac disease. To confirm this diagnosis, an upper endoscopy with biopsy was performed, revealing villous atrophy and crypt hyperplasia in the small intestine, definitive for celiac disease. This finding explained the child's malabsorption issues, correlating with the presence of steatorrhea, and provided a clear direction for dietary modifications.
In parallel, the nutritionist and the rest of the healthcare team initiated a gluten-free diet, which aimed to eliminate gluten-containing foods and focus on nutrient-dense alternatives to address her deficiencies. Early signs of response to this intervention were promising; the child showed a slight increase in energy levels and a growing interest in play activities. However, the team remained cautious, aware that nutritional rehabilitation and recovery would take time, requiring ongoing monitoring and adjustment of the dietary plan.
Despite these positive developments, the team remained vigilant for potential complications. The child's initial presentation of elevated liver enzymes prompted further investigation to rule out additional hepatic pathology. A follow-up liver ultrasound showed mild hepatomegaly, consistent with the inflammatory effects of untreated celiac disease, but no structural abnormalities. The healthcare team discussed the importance of continuous monitoring of liver function tests as part of the child's comprehensive care plan. This case exemplifies the necessity of integrating various clinical insights and maintaining a proactive approach to ensure the child's gradual recovery and developmental progress.
Section 3
As the healthcare team continued to monitor the child's progress on the gluten-free diet, they soon noticed a change in her patient status that warranted further attention. Despite initial improvements in energy levels and physical activity, the child began exhibiting signs of irritability, increased fatigue, and occasional abdominal pain. These symptoms were accompanied by a plateau in weight gain, raising concerns about potential new complications or inadequately addressed nutritional needs.
A comprehensive review of her current intake revealed that while gluten had been successfully eliminated, there might be other dietary deficiencies contributing to her symptoms. The pediatrician ordered a series of blood tests to reassess her nutritional status. The results showed suboptimal levels of iron, vitamin D, and zinc, indicative of ongoing malabsorption issues despite the dietary modifications. Her hemoglobin level had decreased to 10.5 g/dL, suggesting mild anemia, and her vitamin D level was measured at 18 ng/mL, below the recommended threshold. Zinc levels were also marginally low at 60 µg/dL.
In response to these findings, the nutritionist and healthcare team adjusted the child's dietary plan to include gluten-free options fortified with these essential nutrients. Iron supplements and vitamin D were prescribed, and foods rich in zinc, such as lean meats, beans, and seeds, were emphasized. The team also scheduled regular follow-ups to monitor the child's response to these interventions, ensuring her nutritional status improved and optimizing her growth and developmental trajectory. This adjustment highlighted the importance of continual reassessment and adaptation in managing complex cases of failure to thrive, necessitating an integrated, multidisciplinary approach to care.
Section 4
As the healthcare team continued to monitor the child's progress following the dietary adjustments and supplementation, they observed some encouraging signs of improvement. Her energy levels began to stabilize, and her irritability seemed to lessen slightly, suggesting a positive response to the interventions. However, during a routine follow-up visit, the nurse noted that the child's abdominal pain persisted and had become more frequent and severe, particularly after meals. Additionally, a new rash had developed on her elbows and knees, raising concerns about another potential underlying issue.
Given these new symptoms, the pediatrician ordered additional diagnostics, including a comprehensive metabolic panel and a skin biopsy, to explore possible causes such as dermatitis herpetiformis, which is associated with celiac disease. The metabolic panel revealed slightly elevated liver enzymes, with ALT at 45 U/L and AST at 42 U/L, which, while not critically high, suggested a need for further investigation into potential liver involvement or continued malabsorption. The skin biopsy confirmed dermatitis herpetiformis, reinforcing the need for strict adherence to a gluten-free diet and possibly indicating gluten contamination in her current regimen.
These findings prompted the healthcare team to intensify their focus on ensuring complete gluten avoidance, possibly involving a dietitian's review of hidden gluten sources. The new complication of dermatitis herpetiformis emphasized the importance of meticulous dietary management and the necessity for ongoing education for both the family and caregivers. The team also decided to initiate a consultation with a pediatric gastroenterologist to assess the need for further gastrointestinal evaluation, ensuring a comprehensive approach to her care and nutritional management. As they navigated these complexities, the team remained committed to optimizing the child's growth and development while addressing any emerging health concerns.
Section 5
As the healthcare team continued to address the complications associated with the child's diagnosis of dermatitis herpetiformis and potential celiac disease, they closely monitored her response to the gluten-free diet. Despite rigorous efforts to eliminate gluten, the child began to experience additional symptoms, including increased fatigue and intermittent fevers, which were not present in the initial assessments. Concerned about these developments, the pediatrician recommended a series of new diagnostic tests, including a complete blood count (CBC) and a celiac serology panel, to gain more insight into her current condition and to assess for any possible infections or additional autoimmune responses.
The CBC results revealed mild anemia with a hemoglobin level of 10.5 g/dL, slightly below the normal range for her age, indicating possible iron deficiency, which is often seen in conjunction with malabsorption issues like celiac disease. The celiac serology panel showed elevated tissue transglutaminase (tTG) antibodies, confirming the persistence of gluten exposure despite dietary modifications. These findings prompted a reassessment of her dietary plan, leading to a more comprehensive review of potential hidden gluten sources in her environment, such as cross-contamination in food preparation areas at home and school.
In light of these new complications, the healthcare team scheduled an expedited visit with the pediatric gastroenterologist to discuss the potential need for an endoscopy to evaluate the extent of intestinal damage and to adjust her treatment plan accordingly. This coordinated approach aimed to address her anemia through appropriate supplementation and to reinforce the education provided to her family about strict gluten avoidance. The team emphasized the importance of ongoing communication and collaboration among all caregivers to ensure the child's health and well-being, setting the stage for the next steps in her journey towards recovery and growth.