tracheostomy - Nursing Case Study

Pathophysiology

• Primary mechanism: A tracheostomy involves creating a direct airway through an incision in the trachea, bypassing upper airway obstructions and facilitating ventilation. This provides an alternative route for breathing when the oral or nasal passages are compromised.

• Secondary mechanism: The procedure alters normal airway resistance and humidification, which can affect mucociliary function. This change requires diligent humidification and suctioning to maintain airway patency and prevent mucus accumulation.

• Key complication: One moderate complication is tracheal stenosis, where repeated pressure from the tracheostomy tube can cause narrowing of the trachea. This necessitates careful monitoring and tube management to prevent airway compromise.

Patient Profile

Demographics:

45-year-old male, factory worker

History:

• Key past medical history: Chronic obstructive pulmonary disease (COPD), recurrent pneumonia, hypertension

• Current medications: Albuterol inhaler, Lisinopril, Warfarin

• Allergies: Penicillin

Current Presentation:

• Chief complaint: Difficulty breathing and increased secretions around tracheostomy site

• Key symptoms: Shortness of breath, frequent coughing, purulent discharge from tracheostomy, mild fever

• Vital signs: Temperature 38.2°C (100.8°F), heart rate 110 bpm, respiratory rate 24 breaths per minute, blood pressure 145/90 mmHg, SpO2 88% on room air

Section 1

Following the initial presentation, the nursing team conducts a thorough assessment of the patient, focusing on both the tracheostomy site and respiratory function. The tracheostomy site is observed to have significant erythema and swelling, with purulent discharge suggesting a possible infection. The patient is experiencing increased work of breathing, utilizing accessory muscles, and breath sounds are diminished bilaterally with scattered wheezes. Auscultation reveals diminished air entry, indicating potential mucus plugging or partial obstruction.

Blood gas analysis reveals a concerning picture: pH 7.32, partial pressure of carbon dioxide (PaCO2) 58 mmHg, and partial pressure of oxygen (PaO2) 65 mmHg, indicating respiratory acidosis with hypoxemia. A chest X-ray is ordered, showing infiltrates in the lower lobes, raising suspicion of pneumonia, possibly contributing to the tracheostomy site infection. The nurse notes that despite supplemental oxygen, the patient's SpO2 remains suboptimal, hovering around 90%.

In light of these findings, the care team decides to initiate broad-spectrum antibiotics, considering the patient's penicillin allergy, and increases humidified oxygen therapy alongside aggressive suctioning. These interventions aim to manage the infection, improve oxygenation, and alleviate respiratory distress. The team is vigilant for signs of tracheal stenosis or worsening respiratory failure, understanding the delicate balance required given the patient's COPD and current respiratory compromise. As the patient stabilizes, the need for potential bronchoscopy to clear any significant obstructions or assess for further complications is considered, marking the next critical step in the patient's care pathway.

Section 2

As the interventions are implemented, the nursing team closely monitors the patient's response to treatment. Over the next 24 hours, there is a subtle change in the patient's status. The erythema and swelling around the tracheostomy site appear slightly reduced, yet purulent discharge persists, indicating that while the antibiotics are starting to take effect, the infection is still present. The patient's respiratory status shows some improvement; the work of breathing has decreased slightly with less use of accessory muscles, suggesting that the aggressive suctioning and humidified oxygen therapy are helping to clear the airways and improve ventilation.

However, new complications arise as the patient begins to exhibit signs of increasing restlessness and confusion, which could be indicative of worsening hypoxemia or hypercapnia. A repeat blood gas analysis reveals a pH of 7.31, PaCO2 of 60 mmHg, and PaO2 of 63 mmHg, showing that respiratory acidosis and hypoxemia persist, although the parameters have not worsened significantly. The patient's SpO2 remains around 88-90% despite the interventions, indicating ongoing respiratory compromise. Vital signs show a slight increase in heart rate to 105 bpm and a blood pressure of 130/85 mmHg, which may reflect the body's compensatory response to the respiratory distress.

In light of these ongoing challenges and the patient's altered mental status, the clinical team considers the possibility of tracheal stenosis or unresolved mucus plugging that may not have been fully addressed. Given the persistent respiratory acidosis and hypoxemia, a bronchoscopy is now prioritized to evaluate the trachea for any blockages and to ensure that the airways are clear, which is crucial for preventing further respiratory deterioration. This next step is critical in addressing the potential mechanical issues contributing to the patient's compromised respiratory state and in guiding further therapeutic interventions.

Section 3

The bronchoscopy is conducted promptly, revealing partial obstruction of the trachea by thick mucus plugs and mild tracheal stenosis at the site of the tracheostomy. The pulmonologist successfully clears the mucus plugs, resulting in an immediate improvement in the patient's respiratory effort. Post-procedure, the patient is carefully monitored in the intensive care unit, where the nursing team notes a gradual increase in SpO2 to 92-93% with supplemental oxygen, indicating a positive response to the intervention. The patient's breathing becomes less labored, and the use of accessory muscles diminishes further, suggesting improved airway patency and ventilation.

However, the tracheal stenosis requires careful management to prevent further complications. The medical team discusses the potential need for corticosteroids to reduce tracheal inflammation and considers the possibility of surgical intervention if the stenosis does not resolve with conservative measures. Meanwhile, to address the ongoing infection, the antibiotic regimen is reviewed and adjusted based on the latest culture sensitivities, aiming for more targeted antimicrobial therapy.

Throughout the next 12 hours, the patient's mental status begins to improve, with a reduction in restlessness and confusion. This cognitive clarity, coupled with improving blood gas values — pH of 7.35, PaCO2 of 55 mmHg, and PaO2 of 68 mmHg — suggests that the interventions are effectively reducing hypercapnia and hypoxemia. The nursing team continues to emphasize meticulous tracheostomy care, frequent suctioning, and optimal humidification to maintain the airway and prevent further mucus accumulation. With vigilant monitoring and a collaborative approach to management, the care team remains focused on preventing further complications and promoting the patient's recovery trajectory.

Section 4

As the patient continues to recover in the intensive care unit, the nursing team remains vigilant for any signs of new complications. Approximately 24 hours post-bronchoscopy, the patient begins to exhibit subtle signs of respiratory distress, characterized by an increase in respiratory rate to 28 breaths per minute and a slight decrease in SpO2 levels to 90% despite supplemental oxygen. Auscultation reveals diminished breath sounds over the right lower lung field, raising suspicion of atelectasis or possible mucus plugging. The team promptly initiates a chest X-ray, which confirms the presence of right lower lobe atelectasis, likely due to mucus obstruction.

In response to these findings, the care team intensifies pulmonary hygiene measures, including increased frequency of chest physiotherapy and postural drainage, alongside continued suctioning and humidification. The patient's vital signs remain stable otherwise, with a heart rate of 95 beats per minute, blood pressure of 120/75 mmHg, and a temperature of 37.5°C. Arterial blood gas analysis is repeated, showing a pH of 7.34, PaCO2 of 58 mmHg, and PaO2 of 66 mmHg, indicating persistent respiratory acidosis and mild hypoxemia. The medical team decides to initiate a trial of bronchodilators to improve airway clearance and prevent further atelectasis.

As interventions are adjusted, the patient shows signs of gradual improvement. The respiratory rate decreases to 24 breaths per minute, and SpO2 levels stabilize at 92% with continued supplemental oxygen. The nursing team continues to monitor the patient closely, aware that maintaining airway patency and preventing further complications are crucial to the patient’s recovery. The collaborative efforts of the multidisciplinary team highlight the importance of dynamic clinical reasoning and adaptive management strategies in addressing evolving respiratory challenges. The patient’s journey remains a delicate balance between managing underlying conditions and preventing new complications, requiring continuous assessment and timely intervention.

Section 5

As the patient continues to recover, the healthcare team remains vigilant for any new complications. Approximately 48 hours after the initial episode of atelectasis, the patient begins to exhibit changes in mental status, becoming increasingly lethargic and difficult to arouse. The nursing team promptly conducts a neurological assessment, noting that the patient's Glasgow Coma Scale (GCS) score has decreased to 10 from a previous score of 14. Vital signs reveal a slight increase in blood pressure to 130/80 mmHg and a mild tachycardia with a heart rate of 102 beats per minute. The patient's respiratory rate remains stable at 24 breaths per minute, but SpO2 levels have dropped slightly to 89% on supplemental oxygen.

Concerned about these changes, the team orders a repeat arterial blood gas analysis, which reveals a pH of 7.30, PaCO2 of 62 mmHg, and PaO2 of 64 mmHg. This indicates worsening respiratory acidosis and hypoxemia, suggesting that carbon dioxide retention may be contributing to the patient's altered mental state. Additionally, a repeat chest X-ray shows persistent right lower lobe atelectasis despite aggressive pulmonary hygiene measures. Given these findings, the team considers the possibility of a more significant mucus plug or inadequate ventilation as underlying factors.

In response to these developments, the multidisciplinary team decides to escalate the level of respiratory support. The patient is placed on non-invasive positive pressure ventilation (NIPPV) to enhance alveolar ventilation and improve gas exchange. Concurrently, the team increases the frequency and intensity of suctioning and continues administering bronchodilators. As these interventions are implemented, the nursing team closely monitors the patient's neurological status and respiratory parameters, prepared to make further adjustments as necessary. The patient's journey highlights the critical importance of ongoing assessment and the need for timely intervention to manage complications and support recovery.