hemothorax - Nursing Case Study

Pathophysiology

• Primary mechanism: Hemothorax occurs when blood accumulates in the pleural space, often due to trauma which causes rupture of blood vessels in the chest cavity. This leads to compromised lung expansion and impaired gas exchange.

• Secondary mechanism: The presence of blood in the pleural space induces an inflammatory response, which can further exacerbate pleural effusion and lead to fibrotic changes, hindering lung re-expansion.

• Key complication: If not promptly managed, hemothorax can result in hypovolemic shock due to significant blood loss, potentially leading to cardiovascular instability and respiratory failure.

Patient Profile

Demographics:

45-year-old male, construction worker

History:

• Key past medical history: Hypertension, smoking history of 20 pack-years, no prior thoracic surgeries

• Current medications: Lisinopril 10 mg daily

• Allergies: Penicillin

Current Presentation:

• Chief complaint: Shortness of breath and chest pain

• Key symptoms: Dyspnea, pleuritic chest pain, decreased breath sounds on the right side, mild cough with hemoptysis, fatigue

• Vital signs: Blood pressure 100/60 mmHg, heart rate 110 bpm, respiratory rate 28 breaths per minute, temperature 99.1°F, oxygen saturation 88% on room air

Section 1

As the healthcare team continues to monitor the 45-year-old male construction worker admitted with a hemothorax, new diagnostic results have emerged, providing critical insights into his condition. A chest X-ray confirms the presence of a significant right-sided pleural effusion, likely consisting of blood, compressing the right lung and displacing the trachea slightly to the left. A subsequent CT scan of the chest reveals active bleeding from an intercostal artery, contributing to the ongoing accumulation of blood in the pleural space. Laboratory tests show a hemoglobin level of 9 g/dL, indicating anemia likely due to blood loss, and a hematocrit of 28%, further supporting the diagnosis of significant hemorrhagic activity. These findings underscore the urgency of intervention to prevent further deterioration.

In response to the imaging and lab results, the medical team decides to perform a thoracentesis to relieve pressure on the lung and improve respiratory function. Approximately 800 mL of blood is aspirated from the pleural space, providing temporary relief in respiratory symptoms and a slight improvement in oxygen saturation to 91% on supplemental oxygen. However, repeat vital signs show he persists with tachycardia at 108 bpm and hypotension with a blood pressure of 96/58 mmHg, raising concerns about ongoing blood loss and the risk of hypovolemic shock. This prompts consideration for further invasive procedures, such as a chest tube insertion for continuous drainage and potential surgical intervention to address the source of bleeding.

These developments highlight the complexities of managing a hemothorax and the necessity of vigilant monitoring and timely intervention. As the patient's clinical picture evolves, the healthcare team must weigh the risks and benefits of additional diagnostic and therapeutic measures, keeping in mind the patient's pre-existing conditions, such as hypertension, which may complicate his hemodynamic stability. The next steps will focus on stabilizing the patient while planning for definitive management to prevent long-term complications such as fibrothorax, which could impair lung function permanently.

Section 2

As the team continues to assess the patient's response to the thoracentesis and current management plan, new diagnostic results highlight further complications. A follow-up chest X-ray indicates that, despite the removal of blood, there is persistent opacity in the right pleural cavity, suggesting ongoing bleeding or re-accumulation of fluid. The previously noted tracheal deviation has improved slightly, but there remains a significant mediastinal shift, warranting immediate attention. Additionally, an arterial blood gas analysis shows a pH of 7.32, PaCO2 of 52 mmHg, and PaO2 of 65 mmHg, indicating respiratory acidosis and hypoxemia, which could be attributed to impaired gas exchange due to lung compression and evolving respiratory distress.

Clinically, the patient exhibits new signs of distress. He reports increased dyspnea and exhibits accessory muscle use, with respiratory rate escalating to 28 breaths per minute. Auscultation reveals diminished breath sounds on the right side, and there are audible crackles on the left, suggesting compensatory overuse or potential fluid overload secondary to aggressive fluid resuscitation. His mental status begins to fluctuate, with episodes of confusion and restlessness, pointing towards possible cerebral hypoxia. The patient's skin is cool and clammy, and capillary refill is delayed, further indicating compromised perfusion.

In response to these findings, the medical team urgently discusses the need for a chest tube insertion to facilitate continuous drainage and prevent further respiratory compromise. Simultaneously, consideration is given to transferring the patient to the operating room for surgical exploration and repair of the bleeding intercostal artery, especially given the risk of fibrothorax and permanent lung impairment if the bleeding is not controlled. With the patient's hypertension history in mind, the team must carefully balance volume resuscitation efforts to stabilize hemodynamics without exacerbating pulmonary edema. As these plans are set in motion, continuous monitoring and reassessment will be crucial to navigate this complex clinical scenario effectively.

Section 3

As the team proceeds with the chest tube insertion, initial assessment findings post-procedure reveal a partial improvement in the patient's respiratory status. The chest tube drains a significant amount of blood and serous fluid immediately upon insertion, temporarily alleviating the mediastinal shift and slightly enhancing lung expansion. The patient's respiratory rate decreases to 24 breaths per minute, and accessory muscle use reduces, indicating some relief from the respiratory distress. However, despite these initial positive changes, the arterial blood gas analysis remains concerning, with pH improving marginally to 7.34 but PaCO2 persistently elevated at 50 mmHg, and PaO2 at 68 mmHg, suggesting continued respiratory compromise with inadequate ventilation and perfusion.

While the chest tube provides some respite, the patient’s mental status continues to fluctuate, with episodes of confusion persisting. The nursing team notes slight improvement in skin perfusion; however, capillary refill remains delayed at over 3 seconds. The patient remains cool and clammy, indicating ongoing systemic hypoperfusion. Vital signs are closely monitored, with blood pressure readings showing hypotension at 90/60 mmHg, raising concerns about potential hypovolemic shock secondary to ongoing blood loss. This necessitates cautious fluid resuscitation, balancing the risk of exacerbating pulmonary edema while ensuring adequate circulatory volume to maintain organ perfusion.

Given these developments, a repeat chest X-ray is ordered, which shows persistent opacity in the right pleural space, suggesting that the bleeding source may not be adequately controlled by drainage alone. The medical team is now faced with a critical decision: whether to proceed with surgical exploration to definitively address the source of bleeding and prevent further complications such as fibrothorax or respiratory failure. The patient's evolving status, with labile vital signs and fluctuating mental status, underscores the urgency of the situation and the need for decisive action to stabilize him and prevent further deterioration. As discussions continue, the nursing team prepares to support the patient through potential surgical intervention, ensuring that all necessary pre-operative assessments and preparations are completed efficiently.

Section 4

As the medical team deliberates over the necessity of surgical intervention, the patient's condition takes a concerning turn. The nurse notes an increase in the patient's respiratory rate to 28 breaths per minute, accompanied by renewed use of accessory muscles and audible wheezing upon auscultation. This escalation in respiratory distress prompts the team to reassess the chest tube's functionality and position. Despite continuous drainage, there is a suspicion that a clot may be obstructing the tube, limiting its effectiveness in preventing further mediastinal shift and pleural effusion accumulation.

Meanwhile, the patient's blood pressure remains tenuously low, now reading 85/55 mmHg, despite cautious fluid resuscitation efforts. Heart rate has increased to 120 beats per minute, indicating compensatory tachycardia. The patient's skin remains cool and clammy, and capillary refill time is further delayed. Mental status continues to fluctuate, with the patient growing increasingly agitated and uncooperative, complicating ongoing assessments and interventions. A repeat arterial blood gas analysis shows a persistent acidosis with a pH of 7.32, PaCO2 remaining elevated at 52 mmHg, and PaO2 dropping to 64 mmHg, highlighting the inadequate oxygenation and ventilation.

In light of these changes, the medical team decides to expedite preparations for surgical exploration. The potential for ongoing intrathoracic bleeding and the risk of fibrothorax or worsening respiratory failure necessitate urgent action. The nursing team focuses on ensuring the patient is stabilized as much as possible pre-operatively, optimizing oxygen delivery with increased supplemental oxygen, and preparing for potential transfusion to address hypovolemic status. As the patient is prepped for surgery, the multidisciplinary team maintains close communication, ensuring all team members are aligned in their approach to provide the best chance for a positive outcome.

Section 5

As the patient is being prepared for surgery, the team receives new diagnostic results from a recent chest X-ray and CT scan, which confirm the suspicion of a large clot obstructing the chest tube, further complicating the hemothorax. The imaging also reveals a significant mediastinal shift to the left, indicating the potential for compromised cardiac function as well as the possibility of a tension hemothorax developing if the obstruction is not quickly addressed. This underlines the urgency of surgical intervention to evacuate the hemothorax and restore normal pulmonary mechanics.

Additionally, laboratory results return showing a hemoglobin level of 8.5 g/dL, suggesting significant blood loss and reinforcing the need for transfusion as part of the pre-operative management to prevent further hemodynamic instability. The nursing team administers packed red blood cells to improve oxygen-carrying capacity and support the patient’s perfusion status. Despite these efforts, the patient's mental status continues to deteriorate, with increasing confusion and disorientation, likely due to ongoing hypoxia and evolving respiratory acidosis.

The team refocuses their efforts on optimizing the patient's respiratory status, increasing the FiO2 to 60% via a non-rebreather mask, while preparing for possible intubation if the patient's respiratory effort fails to improve. The urgency of the situation is communicated to the surgical team, who prioritize this case to prevent further complications such as fibrothorax or irreversible lung damage. Through coordinated efforts, the team remains committed to stabilizing the patient for a successful surgical outcome, understanding that timely and effective intervention is critical to reversing the current trajectory of decline.