Hip fracture related to ground level fall - Nursing Case Study

Pathophysiology

• Primary mechanism: Osteoporosis weakens bone integrity, reducing the ability to absorb impact forces during a ground level fall, significantly increasing the risk of hip fractures in the elderly population.

• Secondary mechanism: Muscle atrophy and decreased proprioception in older adults impair balance and protective reflexes, contributing to inadequate fall prevention and improper impact distribution, exacerbating fracture risk.

• Key complication: A hip fracture can lead to severe complications such as avascular necrosis due to disrupted blood supply to the femoral head, increasing the risk of joint collapse and necessitating surgical intervention.

Patient Profile

Demographics:

78-year-old female, retired school teacher

History:

• Key past medical history: Osteoporosis, hypertension, type 2 diabetes, chronic kidney disease stage 3

• Current medications: Alendronate, Lisinopril, Metformin, Insulin, Amlodipine

• Allergies: Penicillin

Current Presentation:

• Chief complaint: Severe pain in the right hip and inability to bear weight following a fall

• Key symptoms: Right hip pain, swelling and bruising around the hip, dizziness, confusion, and decreased urine output

• Vital signs: Blood pressure 85/50 mmHg, heart rate 120 bpm, respiratory rate 28 breaths/min, temperature 101.4°F, oxygen saturation 88% on room air

Section 1

Initial Assessment Findings:

Upon arrival at the emergency department, the initial assessment of the 78-year-old female revealed significant clinical concerns. The patient was found to be in acute distress, exhibiting signs of hypovolemic shock likely secondary to internal bleeding from the hip fracture. Her vital signs indicated hypotension with a blood pressure of 85/50 mmHg, tachycardia with a heart rate of 120 bpm, tachypnea with a respiratory rate of 28 breaths/min, and hypoxia with an oxygen saturation of 88% on room air. The elevated temperature of 101.4°F raised suspicion for a potential infectious process. Physical examination of the right hip revealed extensive swelling, bruising, and tenderness, along with an obvious deformity indicating a possible displaced fracture.

The patient's mental status was notably altered; she was confused and disoriented, which could be attributed to a combination of factors including pain, hypoxia, and potential infection. The decreased urine output, coupled with her history of chronic kidney disease stage 3, suggested compromised renal perfusion and function, necessitating urgent intervention. Laboratory tests were promptly ordered, including a complete blood count, metabolic panel, coagulation profile, and blood cultures, to assess for anemia, electrolyte imbalances, renal function deterioration, and infection.

This assessment required immediate initiation of intravenous fluids to stabilize hemodynamics, supplemental oxygen to address hypoxia, and pain management to alleviate discomfort. The complexity of the case was compounded by the need to carefully consider the patient's chronic conditions, medication interactions, and the potential for acute complications such as avascular necrosis or sepsis. The clinical team prepared for urgent imaging studies to confirm the fracture type and extent, while coordinating with orthopedic and nephrology specialists to develop a comprehensive management plan tailored to the patient's multifaceted needs.

Section 2

As the clinical team awaited the results of the laboratory tests and imaging studies, the patient's condition began to deteriorate further, indicating a change in her status. Her level of consciousness decreased, and she became unresponsive to verbal stimuli, requiring a painful stimulus to elicit a minimal response. This decline in neurological status coincided with a further drop in oxygen saturation to 84% despite the administration of supplemental oxygen, suggesting worsening hypoxia. The patient's respiratory rate increased to 32 breaths/min, and her work of breathing became more labored, indicating potential respiratory failure. Additionally, her blood pressure continued to fall, now measuring 78/45 mmHg, and her heart rate remained elevated at 130 bpm, with signs of peripheral cyanosis becoming more pronounced.

The initial laboratory results returned, revealing alarming findings: a hemoglobin level of 7.2 g/dL, indicative of significant anemia likely due to ongoing internal bleeding. Her white blood cell count was elevated at 18,000/mm³, raising further suspicion of an infectious process. The metabolic panel showed worsening renal function, with a creatinine level rising to 3.8 mg/dL and blood urea nitrogen (BUN) at 58 mg/dL, highlighting acute kidney injury superimposed on her chronic kidney disease. Electrolyte imbalances were also noted, with hyponatremia and hyperkalemia that required immediate correction to prevent cardiac complications. The coagulation profile showed an elevated international normalized ratio (INR) of 1.7, increasing the risk of further bleeding.

In light of these findings, the clinical team rapidly escalated their interventions. They initiated a blood transfusion to address the severe anemia and administered broad-spectrum antibiotics to treat the suspected infection. High-flow oxygen therapy was implemented to improve oxygenation, and vasopressor support was considered to stabilize her hemodynamics. The team also consulted with the intensivist and nephrology specialist to discuss the potential need for renal replacement therapy due to the acute deterioration in renal function. Despite these efforts, the patient's condition remained critical, underscoring the need for continued vigilance and dynamic clinical decision-making as they prepared for possible surgical intervention to address the hip fracture and control the bleeding source.

Section 3

As the clinical team continued to monitor the patient closely, new diagnostic results from the CT scan and additional laboratory tests provided further insight into her rapidly evolving condition. The CT scan revealed a significant retroperitoneal hematoma, likely contributing to the ongoing internal bleeding and anemia. This finding explained her worsening hemodynamic instability and necessitated urgent surgical intervention to control the bleeding source. Furthermore, the imaging indicated a high degree of comminution in the hip fracture, complicating potential surgical repair and increasing the risk for post-operative complications.

In parallel, the patient's blood gas analysis showed severe metabolic acidosis, with a pH of 7.20 and bicarbonate level of 12 mEq/L, suggesting lactic acidosis likely from hypoperfusion and sepsis. The lactate level was critically elevated at 6.5 mmol/L, corroborating the suspicion of an underlying septic process. Her electrolyte imbalances persisted, with potassium levels now dangerously elevated at 6.3 mEq/L, raising an immediate risk for life-threatening cardiac arrhythmias. The nephrology team recommended emergent dialysis to address the hyperkalemia, acidosis, and worsening renal function, while the surgical team prepared for an urgent procedure to address the hematoma and stabilize the fracture.

Despite the initiation of these comprehensive interventions, the patient's status remained precarious. Her mental status showed no improvement, and the cyanosis progressed, indicating a potential need for intubation and mechanical ventilation. The multidisciplinary team faced the challenge of balancing the urgent need for surgical intervention with the high risk of perioperative complications, given her critical state. This scenario required advanced clinical reasoning and constant reassessment, as the team anticipated further complications and adjusted their strategies to optimize her chances of recovery.

Section 4

As the surgical team proceeded with the operation to address the retroperitoneal hematoma and stabilize the hip fracture, the patient was closely monitored in the ICU. Despite successful surgical intervention, the post-operative period was fraught with challenges. The patient developed acute respiratory distress, necessitating immediate intubation and mechanical ventilation. Arterial blood gas analysis post-intubation revealed persistent metabolic acidosis with a pH of 7.18 and a declining bicarbonate level of 10 mEq/L, alongside a further increase in lactate to 7.2 mmol/L, indicating unresolved tissue hypoperfusion and possible progression of sepsis. The intensive care team adjusted the ventilator settings to optimize oxygenation and monitored her closely for any signs of acute respiratory distress syndrome (ARDS).

In the hours following surgery, the patient exhibited signs of worsening hemodynamic instability. Her blood pressure dropped to 78/42 mmHg despite aggressive fluid resuscitation and vasopressor support, suggesting refractory shock. Laboratory results indicated a continued rise in inflammatory markers, with a white blood cell count of 18,000/mm³ and a C-reactive protein level of 250 mg/L, further pointing to a systemic inflammatory response. The nephrology team initiated continuous renal replacement therapy (CRRT) to better manage her severe metabolic derangements and prevent further renal deterioration. However, her urine output remained minimal, raising concerns about acute kidney injury.

As the situation evolved, the multidisciplinary team faced the complex task of managing her refractory shock and multi-organ dysfunction. Continuous reassessment of her fluid status, vasopressor requirements, and organ support was crucial. The possibility of an undetected source of infection or a secondary complication, such as abdominal compartment syndrome, was considered. The team planned for an exploratory laparotomy, if necessary, to rule out any additional intra-abdominal pathology. This critical juncture required advanced clinical reasoning to prioritize interventions, reassess strategies, and effectively communicate to optimize the patient's chances of stabilization and recovery.

Section 5

Despite the intensive efforts to stabilize the patient, her condition continued to deteriorate, leading to a new set of critical challenges. In the early hours of the morning, the patient developed a sudden, unrelenting fever, with her temperature spiking to 39.5°C (103.1°F). At the same time, the patient’s heart rate soared to 140 beats per minute, and she became increasingly agitated and difficult to sedate, even on high-dose sedatives and analgesics. These changes prompted an urgent review by the infectious disease team, who suspected a secondary infection. Blood cultures, drawn immediately, subsequently returned positive for Gram-negative bacilli, suggesting a possible ventilator-associated pneumonia or an underlying intra-abdominal infection that had evaded initial detection.

With the new bacteremia finding, the patient's antibiotic regimen was escalated to a broad-spectrum coverage, including meropenem and vancomycin, while awaiting further sensitivity results. The critical care team was faced with the challenge of balancing strong antimicrobial therapy with the potential risk of antibiotic-induced organ toxicity, particularly given the patient’s compromised renal function. Meanwhile, a repeat chest X-ray revealed bilateral pulmonary infiltrates consistent with early ARDS, requiring further fine-tuning of ventilator settings to manage her oxygenation status without exacerbating lung injury. The pulmonary team implemented a lung-protective ventilation strategy, incorporating low tidal volumes and higher positive end-expiratory pressure (PEEP) to improve alveolar recruitment.

As the patient's journey took this complicated turn, the healthcare team keenly focused on reassessment and rapid intervention. Given the complexity of her condition, the priority shifted to maintaining a dynamic approach to monitoring and adjusting her treatment plan. The possibility of performing a CT scan of the abdomen and pelvis was considered to further investigate the potential source of infection or other complications, such as an occult abscess. This strategic decision-making and prioritization of care were crucial as the team endeavored to navigate the evolving landscape of multi-system organ failure, aiming to stabilize and ultimately improve the patient’s precarious health status.