pneumothorax - Nursing Case Study

Pathophysiology

• Primary mechanism: A pneumothorax occurs when air enters the pleural space, the area between the lung and chest wall, disrupting the negative pressure that normally keeps the lung inflated. This can result from trauma, lung disease, or spontaneously due to a rupture in the lung's surface.

• Secondary mechanism: The accumulation of air in the pleural space causes the lung on the affected side to collapse partially or completely, reducing its ability to expand during inhalation. This leads to impaired oxygen exchange and respiratory distress.

• Key complication: If untreated, a tension pneumothorax may develop, where trapped air increases pressure in the chest cavity, causing mediastinal shift and potentially leading to cardiovascular compromise by compressing major blood vessels and reducing cardiac output.

Patient Profile

Demographics:

32-year-old male, construction worker

History:

• Key past medical history: Asthma, smoking history

• Current medications: Albuterol inhaler, Ibuprofen as needed

• Allergies: Penicillin

Current Presentation:

• Chief complaint: Sudden sharp chest pain and difficulty breathing

• Key symptoms: Shortness of breath, decreased breath sounds on the right side, mild cyanosis, anxiety

• Vital signs: Blood pressure 132/86 mmHg, heart rate 112 bpm, respiratory rate 28 breaths per minute, oxygen saturation 88% on room air, temperature 37.2°C

Section 1

As the initial emergency interventions are underway, the patient's status begins to reveal further complications. While receiving high-flow oxygen therapy via a non-rebreather mask, the patient's oxygen saturation improves only slightly to 91%, indicating persistent respiratory compromise. Despite supplemental oxygen, his respiratory rate remains elevated at 30 breaths per minute, and his heart rate climbs to 118 bpm, suggesting increased work of breathing and compensatory mechanisms kicking in. The patient remains visibly anxious and is now exhibiting signs of increasing distress, with accessory muscle use noted on examination.

A chest X-ray is obtained urgently to confirm the suspected pneumothorax and assess its extent. The imaging reveals a large right-sided pneumothorax with significant lung collapse, but more concerning is the subtle shift of the mediastinum towards the left side. This finding raises the suspicion of a developing tension pneumothorax, which correlates with the patient's worsening hemodynamic stability. Concurrently, arterial blood gas analysis reveals a mixed respiratory and metabolic acidosis, with a pH of 7.32, PaCO2 of 50 mmHg, and HCO3- of 20 mEq/L, reflecting impaired ventilation and the beginnings of systemic stress due to inadequate oxygenation.

Recognizing the potential for rapid deterioration, the clinical team prepares for an emergent needle decompression followed by chest tube insertion to alleviate the increasing intrathoracic pressure. The decision to intervene is based on the need to prevent further cardiovascular compromise and to restore normal respiratory mechanics. As these preparations are made, the team must also consider the patient's background of asthma and smoking, which may complicate recovery and necessitate a comprehensive management plan post-decompression to address underlying lung health and prevent recurrence.

Section 2

As the clinical team swiftly proceeds with needle decompression, there is an immediate and audible rush of air, confirming the presence of a tension pneumothorax. This intervention provides partial relief, as evidenced by a slight decrease in the patient's respiratory rate to 26 breaths per minute and a marginal improvement in heart rate to 110 bpm. However, the patient remains tachypneic and anxious, indicating the need for further definitive management. The urgency of the situation requires prompt chest tube placement to ensure continuous decompression and re-expansion of the collapsed lung.

Following the insertion of the chest tube, the patient's oxygen saturation begins to stabilize around 94%, showing gradual improvement. This is a positive sign, yet the patient still exhibits significant work of breathing, with persistent use of accessory muscles and intercostal retractions. Repeat arterial blood gas analysis is performed to monitor changes in his respiratory status. The results show a slight improvement in pH to 7.35, but the PaCO2 remains elevated at 48 mmHg, and HCO3- levels are still low at 21 mEq/L, indicating ongoing respiratory compromise and metabolic acidosis.

Despite these interventions, the patient begins to show signs of a new complication: subcutaneous emphysema, noted as palpable crepitus around the neck and chest wall. This suggests air leakage from the lung into subcutaneous tissue, possibly due to a small bronchopleural fistula. The team recognizes the importance of continuous monitoring and reassessment, ensuring that the chest tube remains patent and effective. A follow-up chest X-ray is ordered to assess lung re-expansion and check for any additional air leaks. Meanwhile, the patient's management plan is adjusted to include close monitoring of vital signs, serial blood gases, and evaluation by a pulmonologist to address the underlying pulmonary issues and reduce the risk of future pneumothorax episodes.

Section 3

As the clinical team continues to manage the patient's condition, a change in patient status emerges, signaling the need for heightened vigilance. Over the next few hours, the patient's respiratory effort appears to increase despite the previously noted partial improvement. The respiratory rate climbs back to 30 breaths per minute, and there is an increased reliance on accessory muscles, suggesting a possible deterioration in respiratory function. The patient's anxiety levels rise, and he becomes increasingly restless, indicating potential worsening of the underlying pulmonary pathology.

A repeat chest X-ray reveals partial re-expansion of the lung; however, there is still evidence of residual pneumothorax and significant subcutaneous emphysema, particularly around the neck and upper chest area. This finding suggests that the air leak might be more persistent than initially anticipated. Furthermore, new diagnostic results from the arterial blood gas analysis show a concerning trend; the pH has slightly decreased to 7.33, and PaCO2 has risen to 52 mmHg, signaling worsening respiratory acidosis. This change in blood gas values indicates inadequate ventilation and possible fatigue of the respiratory muscles.

Recognizing these developments, the clinical team decides to reassess the patency and positioning of the chest tube to ensure optimal drainage. Additionally, the patient is provided supplemental oxygen to maintain adequate oxygenation, and a high-flow nasal cannula is considered to support ventilation. The team plans to consult with a thoracic surgeon to evaluate the need for surgical intervention, such as pleurodesis, if conservative measures fail to resolve the persistent air leak. Continuous monitoring and timely reassessment become critical as the team navigates these new challenges, ensuring the patient remains stable while further diagnostic and therapeutic options are explored.

Section 4

As the clinical team continues to monitor the patient closely, they note a further change in his status. Despite the administered supplemental oxygen and repositioning of the chest tube, the patient’s oxygen saturation levels have dropped to 88% on 6 liters per minute via high-flow nasal cannula, signaling hypoxemia. The patient’s heart rate has increased to 120 beats per minute, and he is now showing signs of diaphoresis, indicating increased physiological stress. His blood pressure remains stable at 130/85 mmHg, but his anxiety and restlessness persist, complicating his ability to maintain effective breathing patterns.

The team decides to conduct a repeat arterial blood gas analysis to better understand the evolving clinical picture. The results show a further decline in pH to 7.30 and an increase in PaCO2 to 58 mmHg, confirming worsening respiratory acidosis and suggesting that the patient's respiratory muscles are continuing to fatigue. Lactate levels have also risen slightly to 2.5 mmol/L, indicating a possible shift towards anaerobic metabolism, likely due to the increased respiratory effort and inadequate oxygen delivery to tissues.

Considering these developments, the team intensifies their focus on ensuring effective ventilation. They deliberate on the potential benefits of non-invasive positive pressure ventilation to alleviate respiratory muscle fatigue and improve gas exchange. Given the persistent air leak and subcutaneous emphysema, the thoracic surgeon is consulted for an urgent evaluation to consider the surgical options, such as pleurodesis or video-assisted thoracoscopic surgery (VATS), to address the underlying pneumothorax more definitively. The team remains vigilant, recognizing that timely intervention is crucial in preventing further deterioration and ensuring the patient’s recovery progresses without additional complications.

Section 5

As the clinical team proceeds with the plan to initiate non-invasive positive pressure ventilation (NIPPV), the patient's response is closely monitored. The application of NIPPV with bilevel settings of inspiratory positive airway pressure (IPAP) at 14 cm H2O and expiratory positive airway pressure (EPAP) at 6 cm H2O results in a gradual improvement of oxygenation, with oxygen saturation levels rising to 92%. The patient’s respiratory rate decreases to 24 breaths per minute, and his heart rate stabilizes at 110 beats per minute, indicating a partial alleviation of respiratory distress. However, the patient remains anxious and reports mild discomfort with the mask, which requires ongoing reassurance and patient education from the nursing staff to ensure compliance and optimize the intervention's effectiveness.

Despite the initial positive response to NIPPV, a new complication arises. The patient's chest tube drainage suddenly increases, with a noticeable change in the character of the fluid to a darker, blood-tinged appearance. This raises concerns about potential hemothorax or damage to surrounding structures. A repeat chest X-ray and CT scan are promptly ordered, revealing an expansion of the pneumothorax and possible hemothorax, with evidence of increasing subcutaneous emphysema. These findings necessitate immediate reassessment by the thoracic surgeon, who determines that surgical intervention is now urgent to prevent further respiratory compromise and correct the underlying issues.

The decision is made to proceed with video-assisted thoracoscopic surgery (VATS) to explore the pleural space, repair any identified air leaks, and evacuate the accumulated blood. The team prepares the patient for surgery, ensuring informed consent and preoperative optimization. Throughout this process, the nursing team remains vigilant, addressing the patient's anxiety and providing support to both the patient and his family, emphasizing the goals of the intervention and the expected outcomes. This crucial step aims to stabilize the patient's condition and set the stage for eventual recovery, minimizing the risk of further complications.