Yung pt. namin sa Community Duty ay may Stroke back 15 years ago. Sa mobility is right hand yung hindi makagalaw pero nakakagalaw nmn paa then, hindi naman apektado face niya kase hindi soya asymmetrical. Gumagamit diya ng tungkod oara sa help niya para makadaan. She can eat, go to cr, change ger lower garments, pero need niya ng assistance sa bathing. Nag wawalk siya sa around ng kanilang bahay as her exercise. Sa speech, nahihirapan talaga dtiya mag salita pero may naiintindihan nmn kmi. Nag undergo siya ng therapy noon kaya medj nagagalaw ang Right. Occupation niya is tailoring before na stroke. This 2025,na afmit siya s ahosp dahil siya difficulty breathing then diagnosed ng Community Acquired Pneumonia than may flyid sa left liver niya then na drain through x-ray guide. May kidney disease din siya maybe because of 15 years of taking meds kaya ganun an yung side effect. May Hypertension siya and namamanage ng meds pero wala namn daw siyang family history of hypertension. Medications: AM: ½ cardiosel (metropolol) PM: Apıxıban 1/2 PM: Amlodipine Morning: ½ Tapazole AM: Apixibon 1/2 Morning. ½ Candez Morning 1/2 Digoxin AM: Febuxostat AM: Renakult - Nursing Case Study
Pathophysiology
• Primary mechanism: Ischemic stroke led to a focal brain injury, primarily affecting the motor cortex controlling the right hand, causing persistent hemiparesis despite therapy.
• Secondary mechanism: Chronic hypertension contributed to vascular damage and increased risk of recurrent stroke, complicating management and rehabilitation efforts.
• Key complication: Long-term medication use, including antihypertensives and anticoagulants, potentially induced chronic kidney disease due to cumulative renal stress, necessitating careful monitoring and dose adjustments.
Patient Profile
Demographics:
45 years old, Female, Former tailor
History:
• Key past medical history: Stroke 15 years ago affecting right hand mobility, Community Acquired Pneumonia, fluid in left lung, kidney disease, hypertension
• Current medications: ½ Cardiosel (Metropolol) AM, Apixaban ½ PM, Amlodipine PM, ½ Tapazole Morning, Apixaban ½ AM, ½ Candez Morning, ½ Digoxin Morning, Febuxostat AM, Renakult AM
• Allergies: None reported
Current Presentation:
• Chief complaint: Difficulty breathing
• Key symptoms: Limited right hand mobility, speech difficulties, assisted bathing, difficulty walking, exercise intolerance
• Vital signs: Critically abnormal (specific values not provided)
Section 1
Initial assessment findings revealed that the patient, a 45-year-old female with a history of stroke and multiple chronic conditions, presented with acute respiratory distress. Vital signs indicated tachypnea with a respiratory rate of 28 breaths per minute, oxygen saturation at 88% on room air, and blood pressure reading of 160/95 mmHg. Auscultation of the lungs revealed diminished breath sounds in the left lower lobe, correlating with previous drainage of fluid from the area. The patient exhibited mild cyanosis and used accessory muscles for breathing, indicating a significant compromise in respiratory function. Neurological assessment showed persistent right hand weakness with a muscle strength graded at 2/5, and speech was slurred but intelligible with effort.
Laboratory results indicated elevated creatinine levels at 2.3 mg/dL, suggesting a worsening renal function, possibly exacerbated by recent medication adjustments or acute illness. Arterial blood gas analysis revealed a mixed respiratory and metabolic acidosis with a pH of 7.30, PaCO2 of 50 mmHg, and HCO3- of 18 mEq/L, indicating both respiratory compromise and renal insufficiency. These findings suggest that the patient’s chronic conditions may be acutely decompensating, requiring immediate intervention to stabilize her respiratory status and renal function.
The clinical team initiated high-flow oxygen therapy, titrating to maintain oxygen saturation above 92%, and adjusted her antihypertensive regimen to address the elevated blood pressure while considering her renal status. A nephrology consult was requested to evaluate the need for further renal support, including potential initiation of dialysis. The nursing team focused on frequent monitoring of vital signs, respiratory status, and neurological function, recognizing the potential for rapid deterioration and the need for continuous reassessment and intervention. This complex clinical picture necessitates a multidisciplinary approach to manage the interplay of her chronic and acute health issues effectively.
Section 2
As the clinical team continued to monitor the patient, a change in her status was noted. Approximately 24 hours after initiating high-flow oxygen therapy, the patient exhibited signs of respiratory fatigue. Her respiratory rate increased to 32 breaths per minute, and despite supplemental oxygen, her oxygen saturation dropped to 85%. She became increasingly lethargic, and her level of consciousness began to fluctuate, raising concerns about hypoxemia and possible hypercapnia.
A repeat arterial blood gas analysis was conducted, revealing a further decline in her respiratory function with a pH of 7.28, PaCO2 of 60 mmHg, and HCO3- of 20 mEq/L. These changes indicated a worsening of respiratory acidosis, likely due to inadequate ventilation. Concurrently, her blood pressure remained elevated at 165/100 mmHg, despite adjustments to her antihypertensive medications, and her renal function continued to deteriorate, with creatinine levels rising to 2.8 mg/dL.
Recognizing the severity of the situation, the medical team decided to escalate respiratory support, considering non-invasive ventilation to improve gas exchange and alleviate respiratory muscle fatigue. A critical care consult was requested to assess the need for more advanced respiratory support or potential intubation. Meanwhile, the nephrology team expedited their evaluation, contemplating early initiation of dialysis to manage her worsening renal failure and mitigate the metabolic acidosis. This situation demanded heightened vigilance from the nursing team, who were tasked with maintaining meticulous monitoring and ensuring timely communication of any further changes in her condition.
Section 3
As the night progressed, the patient's condition continued to evolve, prompting further assessments from the medical team. The nursing staff noted a change in the patient's neurological status; her Glasgow Coma Scale (GCS) score fell from 14 to 10, indicating a deterioration in consciousness. Her pupils were equal and reactive to light, but her response to verbal commands became sluggish, signaling a potential increase in carbon dioxide retention affecting her central nervous system. Concurrently, her vital signs showed a heart rate of 110 beats per minute and a blood pressure of 170/105 mmHg, reflecting ongoing cardiovascular stress despite medication adjustments.
New diagnostic tests, including a chest X-ray and a repeat CT scan of the thorax, were ordered to evaluate the possibility of worsening pneumonia or other complications such as pulmonary embolism or pleural effusion. The chest X-ray revealed an increase in bilateral infiltrates, suggesting the progression of pneumonia, while the CT scan did not show any signs of embolism but confirmed the presence of moderate pleural effusion on the left. In light of these findings, the medical team considered adjusting antibiotic therapy and discussed the potential need for pleural drainage.
In response to her declining renal function, laboratory results indicated a further increase in creatinine to 3.2 mg/dL and a blood urea nitrogen (BUN) level of 48 mg/dL. The nephrology team decided to initiate dialysis in the coming hours to prevent further metabolic derangements and to address the fluid overload contributing to her respiratory difficulties. This decision was crucial as the patient showed signs of fluid retention with mild peripheral edema and crackles on auscultation, indicative of pulmonary congestion. The clinical team remained vigilant, closely monitoring her response to the interventions and ready to adjust the treatment plan as needed based on her evolving condition.
Section 4
As the medical team initiated dialysis, the patient's renal function began to stabilize, with creatinine levels decreasing to 2.8 mg/dL and BUN levels dropping to 42 mg/dL. However, despite this improvement, her respiratory status remained a primary concern. The patient's oxygen saturation fluctuated between 88-90% on 4 liters of nasal cannula oxygen, prompting the team to escalate respiratory support to a high-flow nasal cannula at 40 liters per minute with 60% FiO2. This adjustment improved her oxygen saturation to 93%, yet her work of breathing increased, and she showed signs of respiratory fatigue, including the use of accessory muscles and shallow respirations.
Subsequent arterial blood gas analysis revealed a pH of 7.31, PaCO2 of 55 mmHg, and PaO2 of 58 mmHg, indicating uncompensated respiratory acidosis with hypoxemia. This suggested worsening carbon dioxide retention, likely exacerbated by her underlying pneumonia and pleural effusion. The medical team swiftly reviewed her antibiotic regimen, deciding to broaden coverage to include meropenem and vancomycin, considering the potential for a resistant organism. They also planned for a repeat pleural drainage procedure, suspecting that the effusion could be contributing to her respiratory decline.
As the night continued, the patient's neurological status showed subtle improvement with her GCS score rising to 12, indicating partial responsiveness to the interventions. However, the team remained cautious, aware of the delicate balance needed to manage her complex condition. They prioritized fluid management, carefully monitoring her input and output to prevent fluid overload while ensuring adequate renal perfusion. The focus remained on closely observing her respiratory and neurological responses, ready to make further adjustments to her treatment plan as required, while also preparing for potential new complications that might arise.
Section 5
As the medical team continued monitoring the patient, a change in her status became apparent. During the early morning hours, the patient's respiratory effort increased significantly, and she exhibited more pronounced use of accessory muscles with intercostal retractions. Vital signs showed a heart rate of 110 beats per minute, blood pressure of 140/85 mmHg, respiratory rate of 28 breaths per minute, and an oxygen saturation that had fallen back to 89% despite high-flow nasal cannula support. This prompted the team to reassess her respiratory support strategy urgently.
A repeat chest X-ray was conducted, revealing an increase in the size of the left-sided pleural effusion, suggesting that previous drainage had not been entirely effective. Additionally, laboratory results showed a slight increase in inflammatory markers, with a C-reactive protein (CRP) level of 125 mg/L, indicating ongoing infection or inflammation despite broad-spectrum antibiotic coverage. The patient's white blood cell count remained elevated at 14,500/uL, further corroborating the suspicion of unresolved infection.
In light of these findings, the team decided to proceed with another pleural drainage procedure, this time considering the placement of a chest tube for continuous drainage to better manage the effusion. Concurrently, they contemplated the initiation of non-invasive ventilation to alleviate her respiratory distress and improve gas exchange. The team was mindful of the delicate balance required to manage her fluid status, knowing that any misstep could exacerbate her renal condition or respiratory compromise. As these interventions were planned, they remained vigilant for signs of further complications, understanding the potential for rapid changes in her clinical status.